|Normal||CMR Findings Consistent With Myocardial Inflammation|
|Edema||Lack of evidence for myocardial edema||Patchy areas or regions of high T2 signal intensity indicating focal or regional edema*||Subepicardial or septal layer of high T2 signal intensity indicating regional edema||Transmural high T2 signal intensity indicating regional edema, consistent with but not specific for myocardial inflammation||Global high T2 signal intensity indicating global edema†|
|Hyperemia Capillary leak||Lack of evidence for increased myocardial early gadolinium enhancement ratio||Increased myocardial early gadolinium enhancement ratio‡|
|Irreversible cell injury||Lack of evidence for regional late gadolinium enhancement||Patchy areas of late gadolinium enhancement indicating focal injury||Subepicardial or septal layer of late gadolinium enhancement indicating regional injury||Transmural late gadolinium enhancement, consistent with but not specific for myocardial inflammation|
|Normal||Supportive CMR Findings|
|LV dysfunction||Normal LV function||Regional systolic dysfunction||Global systolic dysfunction|
|Pericardial effusion||Lack of evidence for pericardial effusion||Small pericardial effusion||Moderately large pericardial effusion||Large pericardial effusion without hemodynamic relevance||Large pericardial effusion with hemodynamic relevance|
CMR = cardiovascular magnetic resonance; LV = left ventricular.
↵* To avoid misinterpretation of artifacts, areas with abnormal signal intensity should consist of at least 10 adjacent pixels to be regarded as relevant.
↵† Global high T2 signal is defined by a signal intensity ratio between myocardium and skeletal muscle of ≥2.0.
↵‡ An increased myocardial early gadolinium enhancement ratio is defined by either a signal intensity enhancement ratio between myocardium and skeletal muscle of ≥4.0 or an absolute myocardial enhancement of ≥45%.