Author + information
- Received December 20, 2012
- Accepted January 8, 2013
- Published online June 4, 2013.
- *Department of Medicine, Albert Einstein College of Medicine/Jacobi Medical Center, Bronx, New York
- †Department of Cardiology, Albert Einstein College of Medicine/Montefiore Medical Center, Bronx, New York
- ‡Department of Radiology, Albert Einstein College of Medicine/Jacobi Medical Center, Bronx, New York
- §Department of Medicine (Cardiology), Albert Einstein College of Medicine/Jacobi Medical Center, Bronx, New York
Disturbed calcium-phosphate metabolism can cause metastatic calcium deposition throughout the body. After missing 2 sessions of dialysis and medication noncompliance, severe electrolyte abnormalities developed in our patient (A). On day 1 of admission, a chest computed tomography (CT) scan demonstrated no cardiac or soft-tissue calcifications (B). Concurrently, echocardiography revealed an ejection fraction of 65% by the method of disks with no wall motion abnormalities or evidence of ventricular septal hyperechogeneity (C). On hospital day 9, a repeat CT scan of the chest was performed revealing diffuse amorphous calcifications throughout the left ventricular myocardium and papillary muscles with relative sparing of the interventricular septum (D). A significant increase in radiodensity was detected, consistent with calcium deposition (A). Concomitant echocardiography revealed intact left ventricular function with an ejection fraction of 65% and no wall motion abnormalities with the presence of septal hyperechogenicity. Over the course of her hospitalization, 2 more CT scans were preformed revealing continued evidence of myocardial calcification (A).
- Received December 20, 2012.
- Accepted January 8, 2013.
- American College of Cardiology Foundation